Targeted Peptide Systems

The Compound Library

Every compound in the framework, grouped by the system it actually speaks to — what it does, how it is used, and where it sits on the experience curve. No dosing, and no hype.

How to read this library. Compounds are grouped by the system they act on, not alphabetically, because that is how protocols are actually built. The label on each card — foundational, intermediate or advanced — is about how much margin for error it leaves you, not how well it works.

There are no doses here. That is deliberate. An amount without the context of your situation, your experience and the rest of your protocol is worse than no number at all. Dosing ranges live in Targeted Peptide Systems, and belong in a conversation with a qualified clinician.

If you want this turned into an actual protocol for your situation, the Protocol Builder does it free, and explains its reasoning as it goes.

12 compounds

The growth-hormone axis

These work on the body's own growth-hormone release rather than replacing it. GHRH analogs tell the pituitary to release; GHRPs amplify the pulse when it comes. That distinction matters, because one from each family works together, while two from the same family mostly compete.

Ipamorelin

Selective ghrelin agonist; clean GH pulse
FoundationalGrowth hormone

Cleanest GHRP — minimal cortisol/prolactin bleed. Ideal amplifier alongside a GHRH analog. Best beginner GHRP.

Typical timing: Pre-sleep, 3×/wk pulsatile

Sermorelin

GHRH analog; endogenous GH stimulation
FoundationalGrowth hormone

Gentle physiologic GHRH analog. Best entry GH compound. Pairs synergistically with Ipamorelin (different receptor — not redundant).

Typical timing: Pre-sleep nightly

CJC-1295 (no DAC)

Pulsatile GHRH analog; physiologic GH release
IntermediateGrowth hormone

Amplifies pulsatile GH without blunting natural rhythm. Intermediate upgrade. Combine with one GHRP for pulse amplification.

Typical timing: Pre-sleep, 3×/wk

CJC-1295 (w/ DAC)

Long-acting GHRH analog; sustained GH elevation
IntermediateGrowth hormone

Long-acting GHRH for sustained GH baseline. One GHRH per cycle — do not combine with CJC no-DAC or Sermorelin.

Typical timing: Once weekly SC

GHRP-2

Ghrelin receptor agonist; strong GH pulse
IntermediateGrowth hormone

Potent GH secretagogue. More cortisol/prolactin than Ipamorelin. Intermediate use. One GHRP per cycle only.

Typical timing: 3× daily fasted

GHRP-6

Ghrelin mimetic; GH release + appetite
IntermediateGrowth hormone

Strong appetite stimulation + GH pulse. Best for ectomorphs or hardgainers. One GHRP per cycle.

Typical timing: Pre-workout or pre-sleep

Kisspeptin-10

GnRH stimulation; upstream hormonal regulation
IntermediateLibido Endocrine

Upstream LH/FSH stimulator — supports gonadal axis and testosterone recovery.

Typical timing: Pulsatile 2–3×/wk

MK-677 (Ibutamoren)

Oral ghrelin mimetic; sustained GH/IGF-1
IntermediateGrowth hormone Sleep

Oral GH secretagogue — replaces injection-based GHRPs. Sustains GH/IGF-1 continuously. Monitor fasting glucose; avoid in insulin-resistant endomorphs.

Typical timing: Nightly oral

Tesamorelin

Targeted GHRH analog; GH + metabolic signaling
IntermediateGrowth hormone Fat loss

GHRH analog with visceral fat reduction data. Preferred where GH and metabolic goals overlap, especially in endomorphs.

Typical timing: SC daily AM

Hexarelin

Potent GH secretagogue; high receptor activation
AdvancedGrowth hormone

Strongest GHRP. Rapid receptor desensitisation — 4 wk on/off mandatory. Advanced only.

Typical timing: 2× daily fasted

IGF-1

Direct tissue growth; cellular proliferation
AdvancedGrowth hormone GH / IGF-1

Direct anabolic signalling. Advanced only — not a default addition. One IGF compound per cycle maximum.

Typical timing: Post-training bilateral SC

IGF-1 LR3

Extended IGF-1; enhanced anabolic signaling
AdvancedGrowth hormone GH / IGF-1

Long-acting IGF-1. Greater systemic exposure. One IGF compound per cycle — conflicts with IGF-1.

Typical timing: Post-training SC
8 compounds

Metabolic and fat loss

Appetite, blood sugar and fat storage are one connected system, which is why compounds here overlap so heavily. The incretins dominate the category and are the ones most likely to need a clinician's input.

AOD-9604

Lipolytic signaling independent of GH axis
FoundationalFat loss

GH fragment — lipolysis only, no IGF-1 or GH axis interference. Safe adjunct to any GLP-1 stack.

Typical timing: AM fasted SC, daily

L-Carnitine

Fatty acid transport into mitochondria
FoundationalFat loss Mitochondrial

Fatty acid transport — addresses poor fatty acid oxidation. Complements any metabolic stack.

Typical timing: Pre-workout oral

Lipo-B

Lipotropic B12/MIC; hepatic fat clearance
FoundationalFat loss

Lipotropic blend. Supports hepatic fat clearance. Pairs well with any GLP-1 driver.

Typical timing: Weekly IM

Semaglutide

GLP-1 agonist; metabolic regulation
FoundationalMetabolic Fat loss

Entry-level GLP-1 engine. Best for titration-sensitive beginners. Appetite suppression + glucose regulation. One incretin per stack.

Typical timing: Weekly SC, fixed day

5-Amino-1MQ

NNMT inhibition; enhances NAD+ in fat cells
IntermediateFat loss Mitochondrial

NNMT inhibitor enhancing fat cell metabolism. Complements GLP-1 via a different pathway — not redundant.

Typical timing: Oral with meals

Cagrilintide

Amylin analog; satiety signaling
IntermediateMetabolic

Amylin analogue — satiety via a different receptor than GLP-1. Addresses persistent hunger without receptor redundancy.

Typical timing: Weekly SC

Tirzepatide

GIP/GLP-1 dual agonist; superior modulation
IntermediateMetabolic Fat loss

Dual GIP/GLP-1 agonist — superior fat loss and glycemic control vs single GLP-1. Two receptor pathways.

Typical timing: Weekly SC, fixed day

Retatrutide

GLP-1/GIP/Glucagon triple agonist
AdvancedMetabolic Fat loss

Triple agonist — maximum metabolic driver. Adds glucagon-driven energy expenditure. Reduces need for additional appetite compounds.

Typical timing: Weekly SC, fixed day
6 compounds

Structural repair

Tendon, ligament, gut lining and muscle. The two workhorses act through genuinely different pathways, which is why they are so often discussed together rather than as alternatives.

BPC-157

Angiogenesis + cytoprotection
FoundationalTissue repair Vascular Immune

Broadest-spectrum regenerative peptide. Angiogenesis, tendon, muscle, gut healing. Foundation of any repair stack.

Typical timing: SC near injury site, daily

GHK-Cu

Copper peptide; collagen + matrix remodeling
FoundationalTissue repair Longevity

Copper tripeptide driving collagen synthesis and anti-fibrotic remodelling. Matrix remodeling layer.

Typical timing: SC daily or topical

TB-500

Systemic tissue repair; actin-binding
FoundationalTissue repair Vascular

Distinct cytoskeletal pathway from BPC-157 — highly synergistic pairing. TB-500 or TB-4 per cycle.

Typical timing: SC 2×/wk loading, 1×/wk maint.

ARA-290

Anti-inflammatory + tissue protection
IntermediateVascular Immune Tissue repair

EPO receptor modulator — neuropathic pain, vascular repair, anti-inflammation. Different receptor from BPC-157.

Typical timing: SC 3×/wk

TB-4 (Thymosin β4)

Actin regulation; migration + immune
IntermediateTissue repair Immune

Parent of TB-500 with greater immune modulation. Use TB-500 OR TB-4 per cycle — partially redundant.

Typical timing: SC 2×/wk

B7-33

Relaxin receptor agonist; anti-fibrotic
AdvancedTissue repair Vascular

Anti-fibrotic. Layer after repair initiation for fibrotic conditions. Investigational — advanced use only.

Typical timing: SC daily
7 compounds

Neurocognitive

Focus, memory, mood and sleep. The hardest category to self-assess, because the thing doing the assessing is the thing being changed — which is exactly why written checkpoints matter more here.

DSIP

Delta sleep-inducing peptide
FoundationalSleep

Deepens slow-wave sleep. Pairs with pulsatile GH timing for overnight recovery.

Typical timing: SC 30 min pre-sleep

Oxytocin

Social bonding; parasympathetic + trust
FoundationalSocial Cognition Libido

Social bonding, stress regulation, gut motility. Primary for social dysfunction. Pairs with Selank.

Typical timing: Intranasal situational or daily

Selank

Anxiolytic; GABA modulation + stress
FoundationalCognition Anxiety Sleep

Anxiolytic modulation. Complements Semax (different MOA) — balances stimulation with calm clarity.

Typical timing: Intranasal AM–mid-day

Semax

Nootropic; BDNF upregulation + attention
FoundationalCognition Focus

Foundational cognitive compound — BDNF, attention, focus. AM use only; afternoon dosing disrupts sleep.

Typical timing: Intranasal AM only

Cerebrolysin

Multi-neurotrophic; brain repair
IntermediateCognition Longevity

Multi-neurotrophic — brain repair, cognitive enhancement, neuroprotection. Clinic-level IV administration.

Typical timing: IV infusion 5–10 day course

PE-22-28

Neuroplasticity; antidepressant-like
IntermediateCognition Mood Sleep

Spadin analogue — neuroplasticity, antidepressant signaling, sleep support. Intermediate research compound.

Typical timing: SC AM or PM by use

P-021

Neurogenesis; cognitive preservation
AdvancedCognition Longevity

CNTF-derived neuroprotective peptide. Cognitive preservation. Investigational — advanced only.

Typical timing: SC or intranasal
5 compounds

Immune and inflammatory

Modulators rather than stimulants, in most cases. The general principle in the framework is to settle an over-reactive system before trying to provoke a sluggish one.

Glutathione

Master antioxidant; redox balance
FoundationalImmune Mitochondrial

Master antioxidant. Universal adjunct — no known conflicts. Supports redox balance across all systems.

Typical timing: IV push or nebulized

KPV

Cytokine suppression; anti-inflammatory
FoundationalImmune Tissue repair

Alpha-MSH tripeptide. Converts a repair stack into an anti-inflammatory regenerative stack. Synergistic with BPC-157.

Typical timing: SC or oral daily

Thymosin Alpha-1

T-cell activation; immune regulation
FoundationalImmune

Gold-standard immune modulator. T-cell maturation and innate regulation. Modulate before stimulate.

Typical timing: SC 2×/wk × 4–8 wks

LL-37

Antimicrobial peptide; immune + angiogenesis
IntermediateImmune Vascular

Antimicrobial + angiogenic + anti-inflammatory. Complements Thymosin Alpha-1 via a different pathway.

Typical timing: SC daily or 3×/wk

VIP

Neuroimmune + vascular modulation
IntermediateImmune Cognition

Vasoactive intestinal peptide — neuroimmune regulation. Specialized: chronic inflammatory or neurocognitive patterns.

Typical timing: Intranasal 2×/day
4 compounds

Mitochondrial and cellular energy

Upstream of almost everything else. When fatigue is the presenting complaint and the obvious metabolic causes are ruled out, this is where the framework looks next.

NAD+

Core cellular energy cofactor; DNA repair
FoundationalMitochondrial Longevity Cognition

Universal cellular cofactor. Foundational support across all systems. No conflicts — appropriate at all levels.

Typical timing: IV push or oral liposomal AM

Humanin

Mitochondrial protection; neuroprotective
IntermediateMitochondrial Longevity Cognition

Mitochondrial-derived peptide. Neuroprotective + metabolic + anti-apoptotic. Synergistic with MOTS-c and SS-31.

Typical timing: SC 2–3×/wk

MOTS-c

Mitochondrial signaling; metabolic adaptation
IntermediateMitochondrial Longevity

Mitochondrial-derived peptide. Best for metabolic adaptation and insulin sensitivity (AMPK). Synergistic with NAD+.

Typical timing: SC 2–3×/wk, morning

SS-31 (Elamipretide)

Mitochondrial membrane stabilization
IntermediateMitochondrial Vascular Longevity

Mitochondrial membrane stabiliser. Best for oxidative stress and tissue recovery (heart, skeletal muscle). Synergistic with NAD+ and MOTS-c.

Typical timing: SC daily or 5d/wk
2 compounds

Longevity

The slowest category to show anything you can feel, and the one where expectations most need managing. Judged on markers over long horizons, not on how a given week went.

Epitalon

Telomerase activation; circadian regulation
FoundationalLongevity Sleep Immune

Telomere-supporting tetrapeptide. Anti-aging, circadian, immune modulation. Cycled seasonally.

Typical timing: SC daily × 10–20 day course

FOXO4-DRI

Senolytic; clears senescent cells
AdvancedLongevity

Senolytic — space clearly from continuous mitochondrial peptides. Advanced only.

Typical timing: SC 3×/wk × 4 wks
1 compound

Bone

A small category with a narrow, specific job.

Calcitonin

Calcium balance; bone metabolism
IntermediateBone

Calcium regulation and bone metabolism. Indicated for bone density concerns or calcium dysregulation.

Typical timing: SC or nasal daily
2 compounds

Blends

Pre-combined formulations. Convenient, but they remove your ability to change one variable at a time — and they can silently duplicate something already in your protocol.

GLOW Blend

GHK-Cu + TB-500 + BPC-157
FoundationalTissue repair Vascular Immune

All three repair layers in one protocol. Do not add individual blend components alongside.

Typical timing: Per component schedule

KLOW Blend

GLOW + KPV: regeneration + immune modulation
IntermediateTissue repair Vascular Immune

Adds KPV to convert repair stack into anti-inflammatory regenerative stack. Use GLOW or KLOW — not both.

Typical timing: Per component schedule
Next step

Knowing the compounds is not the same as having a protocol.

Which of these belong together, in what order, and for how long — that is the part the framework exists to answer. Start with the free Builder, or read the book it was built from.

For educational and research purposes only. Nothing here is medical advice, diagnosis, or treatment. Many compounds listed are research peptides that are not approved for these uses. Consult a qualified healthcare professional before acting on any information. Published by NuTide Paradigm.